In response to high amounts of phosphate transport into the cell: a. PhoR is phosphorylated by the phosphate transporter protein Pst b. PhoR binds with PhoB to remove PhoB repression of phosphate regulon genes c. PhoR is exported out of the cell to scavenge phosphate molecules d. PhoU changes conformation to allow PhoR to autophosphorylate e. PhoU keeps PhoR bound to the phosphate transporter protein Pst

Match each medication with its description.

The human body needs energy in order to carry out life processes such as breathing. where does the body get this energy? a. from eating food b. from learning about new things c. from lying in the sun d. from sleeping

Ms. w, a 21-year-old woman, came into a clinic after suffering a deep laceration on her foot while walking barefoot around her yard. the wound was cleaned, sutured, and bandaged, and she was released to return home after receiving tetanus antitoxoid. within 72 hours, the wound area was red and swollen, the suture line was dark in color, and it was accompanied by severe throbbing pain. ms. w had a high fever, her heart felt like it was racing, and she was finding it hard to concentraten even on simple tasks. she returned to the clinic and was immediately taken to the hospital. following lab tests, a diagnosis of acute necrotizing fasciitis was made. discussion questions 1. explain why ms. w. received a tetanus antitoxoid before leaving the hospital. (see chapters 3 and 4, infection and passive immunity.) 2. explain how acute necrotizing fasciitis developed in this case and the pathophysiology involved. (see acute necrotizing fasciitis.) 3. what is the potential outcome for ms. w if antibiotic drugs do not reduce the infection quickly?