If an antigen-presenting cell releases primarily interleukin 2 and interleukin 4, you would expect to see more development of cells, which would trigger a(n) response

If an antigen presenting cell mainly releases interleukin 2 and interleukin 4, it would expect to see further development of proinflammatory cytokines by the cells, which would trigger a response (n) it acquired

Explanation:

The antigen presenting cells phagocytose the antigen (which are generally pathogenic microorganisms) and express a molecule of said antigen on its surface presenting it to the rest of the cells, that is why they bear that name.

But this is not their only function, but they also release humoral factor to attract other cells and promote the immune and inflammatory response, and simultaneously they release anti-inflammatory cytokines to be able to repair or control the tissue damage they generate with the release of free radicals. oxygen or toxics to eliminate noxa.

In this way, the cytokines named above are considered to be the connecting bridge between the assimilation of the antigen and the formation of antibodies (activation of acquired immunity) since they are the ones that activate the lymphocyte lineage and mainly the B lymphocytes that by differentiating into plasmacysts they make specific antibodies.

thrombocytopenia, which is a deficiency in blood platelets, can cause bleeding and bruising in the dermis similar to senile purpura. other possible causes include: exposure to ultraviolet rays and aging skin are common causes of senile purpura. senile purpura may also be a sign of collagen loss in the skin and bones.

there are so many examples for that in different areas, like biology experiment carried out in our lab recently.

here's one link: