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Biology, 28.02.2020 18:53 kendallnowell18

Imagine a mutation that causes a neuron to synthesize and incorporate an abnormally large number of potassium leak channels into its plasma membrane. Assume the neuron is otherwise "typical" (all other channels and pumps are unaffected in terms of function or number, ionic concentration gradients across the cell membrane are "typical" for a mammalian neuron, etc.). What will be true of this neuron?

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