Curare, a poison from a South American shrub, blocks the binding of ACh by muscle cells. What would happen to muscle function if a toxic dose of curare entered the bloodstream?

Paralysis: Action potentials would be impeded at the neuromuscular junction, and the nerve would be unable to trigger the muscle to act
Spasm: The muscle cell will run out of ATP and fluid, becoming hyperexcitable and developing uncontrolled forceful contractions

Seizure: A sudden surge of electrical activity in the brain would cause muscles to stiffen and tremble

No effect: Muscle function is not dependent on the binding of acetylcholine

Ms. w, a 21-year-old woman, came into a clinic after suffering a deep laceration on her foot while walking barefoot around her yard. the wound was cleaned, sutured, and bandaged, and she was released to return home after receiving tetanus antitoxoid. within 72 hours, the wound area was red and swollen, the suture line was dark in color, and it was accompanied by severe throbbing pain. ms. w had a high fever, her heart felt like it was racing, and she was finding it hard to concentraten even on simple tasks. she returned to the clinic and was immediately taken to the hospital. following lab tests, a diagnosis of acute necrotizing fasciitis was made. discussion questions 1. explain why ms. w. received a tetanus antitoxoid before leaving the hospital. (see chapters 3 and 4, infection and passive immunity.) 2. explain how acute necrotizing fasciitis developed in this case and the pathophysiology involved. (see acute necrotizing fasciitis.) 3. what is the potential outcome for ms. w if antibiotic drugs do not reduce the infection quickly?